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.Ventilation and gas exchange during prolonged moderate hypoxia in ponies.
Prolonged (900-1100 min) inhalation of 21% O2 at rest and during three constant work rates (0.47, 0.76, and 1.08 W/kg) increased arterial O2 tension (Pao2) and arterial and mixed venous PO2 and PCO2, but decreased arterial and mixed venous pH. Pao2 decreased linearly (P less than 0.05) from 80 to 50 mm Hg and 70 to 50 mm Hg with increasing work rates, respectively. Cardiac output and right ventricular work rate increased linearly (P less than 0.05) with increasing work rates. Arterial and mixed venous blood lactate concentrations increased (P less than 0.05) from 2.6 +/- 1.0 to 3.6 +/- 1.0 and 3.4 +/- 0.7 mmol/L during exercise. Arterial and mixed venous pH decreased (P less than 0.05) from 7.38 +/- 0.04 and 7.34 +/- 0.06 at rest to 7.32 +/- 0.04 and 7.26 +/- 0.03, respectively, with increasing work rates. Arterial and mixed venous PCO2 and arteriovenous PCO2 gradient decreased (P less than 0.05) during exercise. Arterial and mixed venous base excess decreased (P less than 0.05) from -0.52 +/- 0.49 and -0.96 +/- 0.48 mmol/L at rest to -2.71 +/- 1.11 and -3.29 +/- 0.90, respectively, with increasing work rates. Total arteriovenous CO2 difference increased (P less than 0.05) during exercise. From these data it was concluded that arterial PO2 and Pao2 were depressed and arterial pH was lowered at rest, resulting in right heart failure and decreased blood flow to muscle during hypox

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